Obesity and genetics essay. Link between Obesity and Genetics 2022-11-06

Obesity and genetics essay Rating: 7,3/10 918 reviews

Obesity is a major health concern that affects millions of people around the world. While there are many factors that contribute to obesity, such as diet, lifestyle, and environmental factors, there is also evidence to suggest that genetics plays a role in the development of this condition.

Obesity is defined as having an excess amount of body fat, and is typically measured using the body mass index (BMI), which is calculated by dividing a person's weight in kilograms by their height in meters squared. A BMI of 30 or above is considered obese, while a BMI of 25 to 29.9 is considered overweight.

There are several genetic factors that can contribute to obesity. For example, some people are born with a genetic predisposition to store fat more easily than others. This can make it more difficult for them to maintain a healthy weight, even if they follow a healthy diet and exercise regularly.

There are also specific genetic mutations that have been linked to obesity. One such mutation is in the FTO (fat mass and obesity associated) gene, which has been shown to increase the risk of obesity. Other genetic mutations, such as those in the MC4R (melanocortin 4 receptor) gene, have been linked to an increased risk of obesity as well.

However, it is important to note that genetics is only one factor in the development of obesity. Environmental and lifestyle factors, such as diet and physical activity, also play a significant role. For example, people who eat a diet high in calories and unhealthy fats and do not get enough physical activity are more likely to gain weight and become obese.

There are several ways that genetics can influence obesity, but it is important to remember that genetics is just one piece of the puzzle. A healthy diet and regular exercise are key to maintaining a healthy weight and reducing the risk of obesity. While genetics may make it more difficult for some people to maintain a healthy weight, it is still possible to achieve and maintain a healthy weight through lifestyle changes and proper management.

Free Essay: genetics of obesity

obesity and genetics essay

Regardless of what one may deem the appropriate cause, childhood obesity is becoming a slow but steady killer of children worldwide. The association of genes in obesity and overweight problems has been suggested to be high in both monogenic obesity and polygenic forms. It results from the dealings of an extensive variability of hereditary and ecological factors Hirschhorn, 2005. Changes in leptin gene and the leptin receptor are discussed when obesity caused by genetic issues is presupposed. The epigenome essentially serves as the nexus between nature and nurture; it translates environmental stimuli including nutrition, exposure to toxins, and maybe even our thoughts and feelings into proteins that modulate or alter our gene expression.

Next

Obesity and Genetics (300 Words)

obesity and genetics essay

They are not interested in the role of genetics and tend to mention its role only in a perfunctory manner, pointing out other causes as more severe and complicated ones. Such counseling for educational behavior CHILDHOOD 8 might also be an efficient way to prevent obesity in families with a genetic risk of obesity. It would be beneficial if we could dependably measure the extent to which each of these developments could possibly be up under genetic control in humans. Despite the evidence for this relationship, Odgen 2012 states that parents and children not only share genetic constitution but also share very similar environments, therefore this relationship between child and parental obesity could be contributed to either factor. Finally, these efforts made success stories after effectively managing to treat leptin deficiency children using the recombinant leptin protein. A parent needs to set an example for their children.

Next

Obesity And Genetics

obesity and genetics essay

The overall health of individuals also matter. Furthermore, genetic studies are not without their criticisms. The initial stage of the production of pro-opionmelanocortin starts with the production of the release of the POMC at the hypothalamus. And according to Kleiser et al 2009 , obesity may have several short-term consequences e. However, gene discovery studies for both forms of obesity show that they have shared genetic and biological underpinnings, pointing to a key role for the brain in the control of body weight.

Next

Genetics of Obesity Essay

obesity and genetics essay

The lack of POMC in the body exhibits itself through adrenal failure, altered pigmentation and increment in height onset of obesity Millington, 2007, p. When the energy IN equals more than the energy OUT, the body will eventually gain weight. Accessed on 30 th April from. A genetic predisposition may also be related to appetite control. It is caused when someone is stressed out, depressed or angry. The variation has been identified as the cause for some percentage of population variance in BMI and even a higher number of attributable obesity cases. Hence showing that genes affect weight gain and can cause obesity.


Next

Link between Obesity and Genetics

obesity and genetics essay

Now, there are a variety of factors that fall under the category of poor lifestyle. These minor alleles that have been identified include various polymorphisms which code for isoleucine amino acid instead of valine at position 103 103l and the other that codes for leucine instead of isoleucine at 251L of the receptor protein. We should all be mindful of what we eat and what effect it has on our body. Anything over this average can be attributed with raising the average and therefor in need of correction. These genes are leptin, leptin receptor, melanocortin-4 receptor, pro-opiomelanocortin as well as prohormone convertase genes Bouchard, 2009, p.

Next

The genetics of obesity: from discovery to biology

obesity and genetics essay

Obesity can lead to premature death and even cause Type 2 Diabetes Mellitus. The second is to engage in regular physical exercise. Obesity has a very high relapse rate with most people regaining almost all of their previously lost weight in about 5 years. At present, these technologies have made it possible to study the anatomy of the human genome with improved details and this is expected to yield newer insights into pointing out the exact genetic causes of obesity. There is still a possibility that not all causative genes are already known. In a background that does not support obesity, these individuals would be prone being overweight.

Next

Obesity and Genetics

obesity and genetics essay

The connection between regulation of energy intake and obesity became undoubtful and was discussed further. Polygenic or common obesity and rare, severe, early-onset monogenic obesity are often polarized as distinct diseases. Accessed on 30 th April from Medicinenet. Furthermore, genetic studies are not without their criticisms. Studies conducted by Martin et al 2010, 9 have found that small amounts of alleles of PCSK1 and SNPs are associated in high levels of stimulation of glucose proinsulin levels. Foods that are rich in nutrients, with no added sugars, will help balance the blood sugar level. The fast weight gain in the population is more possible as a result of the hanging environment that supports consumption and discourages usage of energy behaviors that are inadequately similar in temperament with individual hunter-gatherer genes.


Next

Obesity : Obesity And Genetics

obesity and genetics essay

For instance, in Asian populations, it is about 35% while in Indian populations about 50%. Major genetic study identifies clearest link yet to obesity risk. It presupposes the presence of a defect only in one particular gene, which makes people eat more so that they get much more energy than those individuals who do not have such issues. The secondary reasons could be related to our mental and psychological health. Evidence proves that it is associated with numerous leading causes of mortality and morbidity. Level 1 interventions for children and adults resort to population prevention initiatives. Skinny does not equal healthy.

Next